Responses to Merck Comments in Archives of Dermatology

 

In August of 1998 I submitted the paper ``Study of the Food and Drug Administration Files on Propecia

'' to the Archives of Dermatology. It was given three peer reviews, all positive, re-formatted as a

Commentary, and accepted after minor revisions. It finally appeared about a year after submission, in the

March 1999 issue. Subsequently, and without my being informed by the editors, comments from three

Merck associates were published without peer review, in their journal. These authors not only showed a

lack of understanding of statistics, simple logic, and some medical matters but were very personally

denigrating. The reader can find their letters in the August and November 1999 issues. The following

material is my technical response to these comments.

 

A. Keith D. Kaufman Comments ( AOD August 1999 )

 

1) Kaufman is correct in noting that my conclusions were ``based on serum and scalp DHT

measurements we made on balding men treated with finasteride ...''. Those were the data supplied by

Merck to the FDA and on which the approval was based. The data showed the percent change in

testosterone (T) to dihydrotestosterone (DHT), was 46% going from zero dosage to .05 mg and staying

flat to 5 mg. Any physician who has taken a pre-med course on elementary physics, especially one with a

laboratory component, who examines the error bars on the FDA plots that I published, would come to

the same conclusion.

 

2) Kaufman disputes the FDA data by referring to a paper (his ref 3) which is ``in press'', so neither I nor

the FDA could possibly have known about it in April of 1998. He quotes the difference in T to DHT

conversion for 1.0 and 0.2 mg samples as 68.5 +/- 1.4% and 61.2 +/- 1.7%, (a difference of 7.3). These

are not the data given to the FDA. Nor did I get a preprint of these results from Kaufman although he

knew about my interest as the result of our phone calls.

 

3) Kaufman further makes claims that the efficacy was better for the 1.0 mg than for the 0.2 mg dosage

quoting his ref. 4, also ``in press''. That is not data on which the FDA acted.

 

4) The hair count study: The number of persons in the sample were only 101 (1 mg) and 99 (0.2 mg). If

Kaufman bothered to read the material that had been submitted by his company to the FDA, page 23 of

NDA 20-788, he would see that Merck had properly given the data as mean change from baseline: 68.7

+/- 17.3 and 54.9 +/- 17.3. These are the widths of the distributions at the 95% confidence interval. At

the risk of boring the reader, we present in Appendix 1 the standard statistical analysis of the data. Yet

Kaufman suggests in his letter in the Archives ( March 1999 issue) (his references 6 and 7) that a

physicist with over 50 years of publishing data in refereed journals, complete with both ``statistical''' and

``systematic errors'', 1 needs to read a book on statistics.

 

5) Kaufman argues that I should have known that he claimed to have data that proved that 1.0 mg was

superior to .2 mg in hair growth. To show this he gives his reference3 (3 and 4) which he lists as ``in

press''. There is very remarkable use of the calendar in his remarks, a kind of new time-reversal

invariance concept.

 

B. Janet L. Roberts Comments ( AOD August 1999 issue)

 

1) Roberts says ``commentaries are not held to the same rigorous scrutiny and high scientific standard as

peer-reviewed articles" This does injustice to the editors of the AOD since the article was peer-reviewed

by three physicians. The Roberts letter was not peer-reviewed.

 

2) Roberts tries to sell the superiority of the 1mg dose by referring to her reference (1) That reference is

also ``in press''. I do not claim clairvoyance.

 

3) Conclusion: Roberts has introduced a free advertisment for Propecia in the Comments Section. My

article never said Propecia did not grow hair.

 

C. Diane Thiboutot Comments (AOD Nov. 1999)

 

1) Thiboutot says: ``Dr. Frankel insinuates that collaboration occurred between physicians and subjects

as regards questionaires and global assessments.'' Absolutely nothing in my paper does so. The placebo

effect is a remarkable effect connecting mind and body. Although it is routinely used in double blind

studies, the effect is rarely understood in its complexity. It is recommended that interested readers study

the recent book The Placebo Effect edited by Anne Harrington of Harvard Medical School.2

 

Roberts is apparently not familiar with the huge literature on placebo effects or she would be able to

distinguish patient and physician assessments based on a ``Patient Hair Growth Questionaire'' or a

physician ``Investigator Assessment'' vs. real data based on the counting of hairs from photographs. The

former are qualitative opinion polls and are usually only used when real measurements of hair counts are

not available. This was not the case here. What Roberts has not understood is that one placebo effect

arises because the patient would like to see the treatment grow hair. So would the physician. To deny this

effect for physicians makes an unflattering statement suggesting that physicians, unlike their patients, do

not have the same human responses. I thought it was worth mentioning, from my examination of the data,

that there was nice evidence here for the placebo-like overestimate of the hair growth for both types of

observers. None of the FDA referees commented on this result.

 

2) Roberts goes into well known conditions on pricing that are irrelevant to the drug dosage approval and

which are well known. I should not have made the remark about solubility in ethanol since the FDA

actually blacked-out, i.e. censored, the actual solubility. I apologize for not telling the dermatologist

readers that there is a trivial way to subdivide the pill: Crush it between two spoons and transfer the very

fine powder to a small clear glass bottle of water. (The containing coating is easy to remove.) An

extremely fine suspension is obtained. Shaking the bottle and removing a tablespoon of liquid is the simple

way to reduce the dose.

 

I cannot understand why saving money ``cannot be condoned'' when the cost is approximately $1000 per

year, not covered by insurance, and the drug must be taken forever in order to preserve any hair growth.

There are no data proving that the lower dosages are ineffective while the physical T to DHT conversion

measurements are flat from .05 to 5 mg. Considering the cost to take the drug for the rest of one's life and

that the drug is too expensive for most young men, an intelligent user might want to do a few month trial at

lower dosage. Nothing to lose but some fuzz.

 

A popular article dealing with my interactions with the FDA, Merck, and medical journals concerning the

finasteride, will appear shortly.

 

propcrapmerck printed May 2, 2000

 

 

Footnotes:

 

1 Apparently the medical profession almost never assigns systematic errors in their presentations and

conclusions, yet in many experiments the systematic errors are larger than the statistical errors.

 

2 ``The Placebo Effect'', Harvard University Press, second printing, 1999